![]() “Angioscintigraphie cardiaque au cours de l’intoxication aïgue par l’oxyde de carbone”. 1976, 38: 712–717.Įlkharrat D, Raphael JC, Tainturier C, Brunel D, De Truchis P, Goulon M. “Echocardiographic findings after acute carbon monoxide poisoning”. “Muscle maximal 02 uptake at constant 02 delivery with and without CO in the blood”. Hogan M, Bebout D, Gray P, Wagner J, West J, Haab P. “02 delivery to contracting muscle during hypoxic or CO hypoxia”. American Physiological Society, Bethesda, 1987, PP 439–456. In Fishman A led) Handbook of physiology of the respiratory system, vol 4: gas exchange”. “Myoglobin 0, tension determined from measurements of carbomyoglobin in skeletal muscle”. “The relative effects of hypoxic hypoxia and carbon monoxide on brain function in rabbits”. Ludbrook G.L., Helps S.C., Gorman D.F., Reilly P.L., North J.B., Grant C. “Mechanism of the toxic action of carbon monoxide”. “Effects of metabolism and distribution of carbon monoxide on blood and body stores”. “The effect of carbon monoxide on the oxyhemoglobin dissociation curve”. “The relation of the action of carbonic oxide to oxygen tension”. “Formulation of the principal factors affecting the rate of uptake of carbon monoxide by normal man”. “Carbon monoxide poisoning, a diagnosis frequently overlooked”. “Prospective study in carbon monoxide intoxication by gas water heaters”. ![]() Well designed prevention programs are urgently needed in our countries to decrease the incidence and consequences of CO poisoning Keywordsįaure J, Arsac P, Chalandre P. It is also indicated for pregnant women after exposure to CO. HBO is recommended for all patients remaining comatose on hospital admission those who have lost consciousness during toxic exposure those with persisting neurological disorders. Oxygen is the basis of the treatment and Hyperbaric oxygen therapy (HBO) has been proven more effective in preventing cognitive sequelae than normobaric oxygen. However, long term neurological manifestations may occur and lead to severe functional impairment and disabilities. Moreover, in addition to the hypoxic injury, the reoxygenation phase may itself induce toxic effects by mechanisms closely related to the ischemia-reperfusion phenomenon.Ĭlinical manifestations include neurological disturbances, cardiac arrhythmias, and respiratory and circulatory failure which usually ceases when the patient is removed from the toxic atmosphere and given oxygen. Beyond the well-known effects of CO on hemoglobin, the role of CO binding to other hemoproteins like myoglobin and cytochrome a-a3 has been identified more recently. Carbon monoxide (CO) poisoning still remains a serious public health problem in Europe.
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